DNP(2 4-Dinitrophenol)CAS 51-28-5 Pharmaceutical Raw Material
Losing Weight Steroids
DNP / 2, 4-Dinitrophenol
Sensitive: Light Sensitive
Appearance: Light yellow to yellow crystals. Soluble in ethanol,
benzene, chloroform and ether, dissolved in lye, slightly soluble
in cold water.
Usage: Weight loss drug.
Packing: foil bag or tin.
Payment: Bank Transfer, Western Union, Money Gram
Express: EMS, DHL, FedEx, etc(door-to-door)
Delivery: Safe & timely, around 1-3 days after payment
2,4-Dinitrophenol is used in the manufacture of dyes, wood
preservatives, and as a pesticide. The acute (short-term) effects
of 2,4-dinitrophenol in humans through oral exposure are nausea,
vomiting, sweating, dizziness, headaches, and loss of weight.
Chronic (long-term) oral exposure to 2,4-dinitrophenol in humans
has resulted in the formation of cataracts and skin lesions, weight
loss, and has caused effects on the bone marrow, central nervous
system (CNS), and cardiovascular system. Limited or no information
is available on the developmental, reproductive, or carcinogenic
effects of 2,4-dinitrophenol in humans. EPA has not classified
2,4-dinitrophenol for carcinogenicity.
2,4-Dinitrophenol (2,4-DNP, or simply DNP) is an organic compound
with the formula HOC6H3(NO2)2. It is a yellow, crystalline solid
that has a sweet, musty odor. It sublimes, is volatile with steam,
and is soluble in most organic solvents as well as aqueous alkaline
solutions. It is a precursor to other chemicals and is
biochemically active, inhibiting energy (ATP) production in cells
with mitochondria and was once used as a diet aid. It is produced
by hydrolysis of 2,4-dinitrochlorobenzene.
DNP was used extensively in diet pills from 1933 to 1938 after
Cutting and Tainter at Stanford University made their first report
on the drug's ability to greatly increase metabolic rate. After
only its first year on the market Tainter estimated that probably
at least 100,000 persons had been treated with DNP in the United
States, in addition to many others abroad. DNP acts as a
protonophore, allowing protons to leak across the inner
mitochondrial membrane and thus bypass ATP synthase. This makes ATP
energy production less efficient. In effect, part of the energy
that is normally produced from cellular respiration is wasted as
heat. The inefficiency is proportional to the dose of DNP that is
taken. As the dose increases and energy production is made more
inefficient, metabolic rate increases (and more fat is burned) in
order to compensate for the inefficiency and meet energy demands.
DNP is probably the best known agent for uncoupling oxidative
phosphorylation. The production or "phosphorylation" of ATP by ATP
synthase gets disconnected or "uncoupled" from oxidation.
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